Phosphaturia is used to describe an impaired capacity to reabsorb phosphate from the glomerular filtrate, leading to urinary phosphate wasting. Reabsorption of phosphate is a saturable carrier-mediated process, and the amount of phosphate excreted in urine varies according to the filtered load. This in turn largely reflects dietary intake, which varies widely. On high intakes, normal individuals excrete large amounts of phosphate as an appropriate adaptive response. On the other hand, phosphaturic individuals with low dietary intake will excrete normal amounts of phosphate
High phosphate/phosphorous levels in urine may mean:
Most people with high phosphate levels don't have symptoms. In some people with chronic kidney disease, high phosphate levels cause calcium levels in the blood to drop. Symptoms of low calcium include:
Senna: --Specific gravity and density increased; hyperazoturia, oxaluria, phosphaturia, and acetonuria. Where the system is broken down, bowels constipated, muscular weakness, and waste of nitrogenous materials, Senna will act as a tonic.
Phosphoric acid: Micturition, preceded by anxiety and followed by burning. Frequent urination at night. Phosphaturia. Weak. Tearing pains in joints, bones, and periosteum. Cramps in upper arms and wrists. Great debility. Pains at night, as if bones were scraped. Stumbles easily and makes missteps.
Kali Phosphoricum: . Very yellow urine. Paralytic lameness in back and extremities. Exertion aggravates. Pains, with depression, and subsequent exhaustion.
Alfalfa: -Kidneys inactive; frequent urging to urinate. Polyuria (Phos ac). Increased elimination of urea, indican and phosphates.
Thalaspi Bursa: Frequent desire; urine heavy, phosphatic. Chronic cystitis. Dysuria and spasmodic retention. Hæmaturia. Accumulation of gravel. Renal colic. Brick-dust sediment. Urethritis; urine runs away in little jets. Often replaces the use of the catheter.